SCMR Level I Course Lithuania 2025-Session 3

Video Transcription

Video Summary

The transcript covers three CMR-focused lectures.<br /><br />First, Associate Professor Thomas Remba reviewed CMR in infiltrative cardiomyopathies: amyloidosis, sarcoidosis, Fabry disease, and hemochromatosis. He explained how cine imaging, late gadolinium enhancement, native T1/T2 mapping, T2*, and extracellular volume (ECV) help identify abnormal myocardial deposition, edema, fibrosis, and iron overload. Key patterns included diffuse wall thickening and difficulty nulling the myocardium in amyloidosis, patchy non-ischemic late enhancement in sarcoidosis, low native T1 in Fabry disease, and low T2* in hemochromatosis. He emphasized correlating CMR findings with clinical history, ECG, and labs.<br /><br />Second, a lecture on myocarditis described how CMR detects myocardial inflammation through edema, hyperemia/capillary leak, and necrosis/fibrosis. The original Lake Louise criteria required two of three findings, while the updated 2018 criteria incorporate T1/T2 mapping and ECV. The speaker highlighted that CMR is especially useful early in the disease course, ideally within the first week, and is now strongly recommended in recent guidelines for diagnosis and follow-up.<br /><br />Finally, the pericardial imaging talk reviewed pericardial anatomy, effusions, pericarditis, constriction, congenital absence of the pericardium, and pericardial cysts. It stressed the value of cine, T1/T2-weighted imaging, LGE, and real-time sequences for detecting inflammation, effusion, and constrictive physiology.

Keywords

CMR

cardiomyopathies

amyloidosis

sarcoidosis

myocarditis

Lake Louise criteria

T1/T2 mapping

pericarditis

pericardial imaging